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dc.contributor.authorMäki-Marttunen, Tuomo
dc.contributor.authorHalnes, Geir
dc.contributor.authorDevor, Anna
dc.contributor.authorWitoelar, Aree
dc.contributor.authorBettella, Francesco
dc.contributor.authorDjurovic, Srdjan
dc.contributor.authorWang, Yunpeng
dc.contributor.authorEinevoll, Gaute T
dc.contributor.authorAndreassen, Ole A
dc.contributor.authorDale, Anders M
dc.date.accessioned2016-05-24T05:09:14Z
dc.date.available2016-05-24T05:09:14Z
dc.date.issued2015
dc.identifier.citationBMC Neuroscience. 2015 Dec 18;16(Suppl 1):P45
dc.identifier.urihttp://hdl.handle.net/10852/50362
dc.language.isoeng
dc.rightsMäki-Marttunen et al.
dc.rightsAttribution 4.0 International
dc.rights.urihttps://creativecommons.org/licenses/by/4.0/
dc.titleTowards "biophysical psychiatry": a modeling approach for studying effects of schizophrenia-linked genes on single-neuron excitability
dc.typeJournal article
dc.date.updated2016-05-24T05:09:14Z
dc.creator.authorMäki-Marttunen, Tuomo
dc.creator.authorHalnes, Geir
dc.creator.authorDevor, Anna
dc.creator.authorWitoelar, Aree
dc.creator.authorBettella, Francesco
dc.creator.authorDjurovic, Srdjan
dc.creator.authorWang, Yunpeng
dc.creator.authorEinevoll, Gaute T
dc.creator.authorAndreassen, Ole A
dc.creator.authorDale, Anders M
dc.identifier.doihttp://dx.doi.org/10.1186/1471-2202-16-S1-P45
dc.identifier.urnURN:NBN:no-53984
dc.type.documentTidsskriftartikkel
dc.type.peerreviewedPeer reviewed
dc.identifier.fulltextFulltext https://www.duo.uio.no/bitstream/handle/10852/50362/1/12868_2015_Article_3909.pdf
dc.type.versionPublishedVersion
cristin.articleidP45


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