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dc.contributor.authorMeier, Silja
dc.contributor.authorAndressen, Kjetil W
dc.contributor.authorAronsen, Jan M
dc.contributor.authorSjaastad, Ivar
dc.contributor.authorSkomedal, Tor
dc.contributor.authorOsnes, Jan-Bjørn
dc.contributor.authorQvigstad, Eirik
dc.contributor.authorLevy, Finn O
dc.contributor.authorMoltzau, Lise R
dc.date.accessioned2016-03-22T05:14:45Z
dc.date.available2016-03-22T05:14:45Z
dc.date.issued2015
dc.identifier.citationBMC Pharmacology and Toxicology. 2015 Sep 02;16(Suppl 1):A66
dc.identifier.urihttp://hdl.handle.net/10852/49980
dc.language.isoeng
dc.rightsMeier et al.
dc.rightsAttribution 4.0 International
dc.rights.urihttps://creativecommons.org/licenses/by/4.0/
dc.titleEnhancement of cAMP-mediated inotropic responses by CNP is regulated differently by PDE2 in normal and failing hearts
dc.typeJournal article
dc.date.updated2016-03-22T05:14:45Z
dc.creator.authorMeier, Silja
dc.creator.authorAndressen, Kjetil W
dc.creator.authorAronsen, Jan M
dc.creator.authorSjaastad, Ivar
dc.creator.authorSkomedal, Tor
dc.creator.authorOsnes, Jan-Bjørn
dc.creator.authorQvigstad, Eirik
dc.creator.authorLevy, Finn O
dc.creator.authorMoltzau, Lise R
dc.identifier.doihttp://dx.doi.org/10.1186/2050-6511-16-S1-A66
dc.identifier.urnURN:NBN:no-53683
dc.type.documentTidsskriftartikkel
dc.type.peerreviewedPeer reviewed
dc.identifier.fulltextFulltext https://www.duo.uio.no/bitstream/handle/10852/49980/1/40360_2015_Article_435.pdf
dc.type.versionPublishedVersion
cristin.articleidA66


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