Syndecan-4 in Cardiac Fibroblasts. Role in Regulation of Extracellular Matrix and Myocardial Stiffness
Metadata
Show metadataAppears in the following Collection
- Institutt for klinisk medisin [10814]
Abstract
No abstract.List of papers
Paper 1. Syndecan-4 signalling via NFAT regulates extracellular matrix production and cardiac myofibroblast differentiation in response to mechanical stress. Herum KM, Lunde IG, Skrbic B, Florholmen G, Behmen D, Sjaastad I, Cathrine R. Carlson, Maria F. Gomez, Geir Christensen. J Mol Cell Cardiol. 2013;54:73-81. The paper is removed from the thesis in DUO due to publisher restrictions. The published version is available at: https://doi.org/10.1016/j.yjmcc.2012.11.006 |
Paper 2. Syndecan-4 is a key determinant of collagen cross-linking and passive myocardial stiffness in the pressure-overloaded heart. Herum KM, Lunde IG, Skrbic B, Louch WE, Hasic A, Boye S, Unger A, Brorson SH, Sjaastad I, Tønnessen T, LinkeWA, Gomez MF, Christensen G. Cardiovasc Res. 2015 May 1;106(2):217-26. Submitted version, the published version is available at: https://doi.org/10.1093/cvr/cvv002 |
Paper 3. Innate immune signalling induces expression and shedding of the heparan sulfate proteoglycan syndecan-4 in cardiac fibroblasts and myocytes, affecting inflammation in the pressure-overloaded heart. Strand ME, Herum KM, Rana ZA, Skrbic B, Askevold ET, Dahl CP, Vistnes M, Hasic A, Kvaløy H, Sjaastad I, Carlson CR, Tønnessen T, Gullestad L, Christensen G and Lunde IG. FEBS J. 2013;280:2228-2247. The paper is removed from the thesis in DUO due to publisher restrictions. The published version is available at: https://doi.org/10.1111/febs.12161 |